HIV-1 and immunopathogenesis [electronic resource] : innate immunity / Luis J. Montaner.

By: Montaner, Luis J [spk]Material type: FilmFilmSeries: Henry Stewart talksBiomedical & life sciences collection. Innate immunity : host recognition and response in health and disease: Publisher: London : Henry Stewart Talks, 2009Description: 1 online resource (1 streaming video file (56 min.) : color, sound)Subject(s): HIV-1 -- immunology | HIV-1 -- pathogenicity | Immunity, InnateOnline resources: Click here to access online | Series
Contents:
Contents: Viral life cycle -- Acute infections -- Immune response -- Timeline to effectors -- Stages of immunopathogenesis -- Macrophages and HIV-1 infection -- Monocyte apoptosis -- Human DC subsets -- Model of DC migration and maturation -- MHC I antigen presentation -- Proposed pathways of human DC development -- C-type lectins -- Nature killer cells -- Natural killer cell recognition of target cells -- Role of HLA and KIR alleles in rate of progression to AIDS -- Measuring recovery following immune reconstitution and HIV-1 therapy -- ART and innate reconstitution -- Model of immune reconstitution timeline -- Does the innate immune activation response determine HIV-1 disease? -- Disease models: it's the activation.
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Animated audio-visual presentation with synchronized narration.

Title from title frames.

Contents: Viral life cycle -- Acute infections -- Immune response -- Timeline to effectors -- Stages of immunopathogenesis -- Macrophages and HIV-1 infection -- Monocyte apoptosis -- Human DC subsets -- Model of DC migration and maturation -- MHC I antigen presentation -- Proposed pathways of human DC development -- C-type lectins -- Nature killer cells -- Natural killer cell recognition of target cells -- Role of HLA and KIR alleles in rate of progression to AIDS -- Measuring recovery following immune reconstitution and HIV-1 therapy -- ART and innate reconstitution -- Model of immune reconstitution timeline -- Does the innate immune activation response determine HIV-1 disease? -- Disease models: it's the activation.

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