Metabolic and biochemical alterations in Alzheimer's disease (Record no. 77535)

000 -LEADER
fixed length control field 02128ngm a2200325 a 4500
001 - CONTROL NUMBER
control field HST2816_1_3
003 - CONTROL NUMBER IDENTIFIER
control field UkLoHST
006 - FIXED-LENGTH DATA ELEMENTS--ADDITIONAL MATERIAL CHARACTERISTICS
fixed length control field m c
007 - PHYSICAL DESCRIPTION FIXED FIELD--GENERAL INFORMATION
fixed length control field cr|cna|||a||||
007 - PHYSICAL DESCRIPTION FIXED FIELD--GENERAL INFORMATION
fixed length control field vz|czazum
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
fixed length control field 110715s2011 enk|||||||||||s|||v|eng d
028 50 - PUBLISHER NUMBER
Publisher number 2816
Source Henry Stewart Talks
035 ## - SYSTEM CONTROL NUMBER
System control number (UkLoHST)2025
040 ## - CATALOGING SOURCE
Original cataloging agency UkLoHST
Language of cataloging eng
Transcribing agency UkLoHST
100 1# - MAIN ENTRY--PERSONAL NAME
Personal name Smith, Mark A.,
Affiliation (Case Western Reserve University, USA)
Relator code spk
245 10 - TITLE STATEMENT
Title Metabolic and biochemical alterations in Alzheimer's disease
Medium [electronic resource] :
Remainder of title facts and fictions /
Statement of responsibility, etc. Mark A. Smith.
260 ## - PUBLICATION, DISTRIBUTION, ETC. (IMPRINT)
Place of publication, distribution, etc. London :
Name of publisher, distributor, etc. Henry Stewart Talks,
Date of publication, distribution, etc. 2011.
300 ## - PHYSICAL DESCRIPTION
Extent 1 online resource (1 streaming video file (51 min.) :
Other physical details color, sound).
490 1# - SERIES STATEMENT
Series statement Alzheimer's disease : neurobiology, pathology and treatment,
International Standard Serial Number 2056-452X
500 ## - GENERAL NOTE
General note Animated audio-visual presentation with synchronized narration.
500 ## - GENERAL NOTE
General note Title from title frames.
505 0# - FORMATTED CONTENTS NOTE
Formatted contents note Contents: What causes selective neuronal loss in AD? -- Tauist and BAPtist hypothesis of AD -- Pathology and oxidative stress relationship -- NFT and amyloid-beta -- Stress-activated protein kinases and phospho-tau -- Amyloid-targeted approaches are unsuccessful -- Amyloid-beta controls redox activity -- Genetic factors are related to oxidative stress -- Therapeutics -- Potential mechanism of ROS generation -- Oxidative modifications and macromolecules -- Oxidative damage directly correlates with mtDNA -- Abnormal mitochondrial distribution in AD brains -- Iron histochemistry -- Antioxidant diet is protective -- Only one non-amyloid drug in phase III: 2010 -- Oxidative stress is necessary but not sufficient -- CaMKII-MYC mice -- Oxidative stress and cell cycle abnormalities -- Early stages of AD disease signals.
506 ## - RESTRICTIONS ON ACCESS NOTE
Terms governing access Access restricted to subscribers.
538 ## - SYSTEM DETAILS NOTE
System details note Mode of access: World Wide Web.
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name as entry element Alzheimer's disease.
650 #2 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name as entry element Alzheimer Disease
General subdivision metabolism.
830 #0 - SERIES ADDED ENTRY--UNIFORM TITLE
Uniform title Henry Stewart talks.
Name of part/section of a work Biomedical & life sciences collection.
-- Alzheimer's disease.
856 40 - ELECTRONIC LOCATION AND ACCESS
Uniform Resource Identifier <a href="https://hstalks.com/bs/2025/">https://hstalks.com/bs/2025/</a>
856 42 - ELECTRONIC LOCATION AND ACCESS
Uniform Resource Identifier <a href="https://hstalks.com/bs/p/529/">https://hstalks.com/bs/p/529/</a>
Materials specified Series

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